tumor necrosis factor-α-308 g/a polymorphisms and risk of hepatocellular carcinoma: a meta-analysis

نویسندگان

soheil tavakolpour iran university of medical sciences, tehran, ir iran; infectious diseases and tropical medicine research center, shahid beheshti university of medical sciences, tehran, ir iran

shahnaz sali infectious diseases and tropical medicine research center, shahid beheshti university of medical sciences, tehran, ir iran; infectious diseases and tropical medicine research center, shahid beheshti university of medical sciences, tehran, ir iran. tel: +98-9123067784, fax: +98-2122546026

چکیده

conclusions this meta-analysis indicated that the tnf-α-308 g/a polymorphism is significantly associated with increased susceptibility to hcc. however, to confirm this finding, more studies are needed on tnf-α-308 g/a polymorphisms associated with hcc. context hepatocellular carcinoma (hcc) is a common disorder throughout the world that can develop due to various factors, including genetics. tumor necrosis factor-α (tnf-α) is the most frequently studied cytokine related to the risk of developing hcc, and an association between the 308 position of the tnf-α promoter (tnf-α-308) and hcc risk has been confirmed in various reports. evidence acquisition the pubmed, scopus, and google scholar databases were searched through july 12, 2015, for studies on associations between tnf-α-308 and the risk of hcc. to determine this association, odds ratios (ors) and 95% confidence intervals (95% cis) were calculated. results a total of 23 case-control studies were investigated, involving 3,389 cases and 4,235 controls. the overall conclusion was that the a allele was more frequent in case groups compared to control groups (13.4% vs. 8.4%). thus, the a allele was significantly associated with increased hcc risk (or = 1.77; 95% ci = [1.26-2.50]; p value < 0.002). in addition to the allelic model, the dominant model (aa + ag vs. gg) was significantly associated with hcc risk (or = 1.80; ci = [1.29-2.51]; p value < 0.001). in the sensitivity analysis for co-dominant (aa vs. gg) and recessive models (aa vs. ag + gg), no trustworthy associations with the risk of hcc development were observed.

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عنوان ژورنال:
hepatitis monthly

جلد ۱۶، شماره ۴، صفحات ۰-۰

کلمات کلیدی
[ ' t u m o r n e c r o s i s f a c t o r ' , ' α ' , ' h e p a t o c e l l u l a r c a r c i n o m a ' , ' p o l y m o r p h i s m s ' , ' t n f ' , ' α ' , 3 0 8 , ' m e t a ' , ' a n a l y s i s ' ]

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